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Dysfunction of tight junctions is thought to be a primary driver of inflammatory bowel disease and has even been tied to enhanced wheat gluten allergies in celiac disease

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These studies suggest that most glutamine-dependent protein translation activities can still proceed under asparagine supplementation in a glutamine-deprived environment, although the exact mechanism is still unknown

Concurrent delivery of a Trp2 peptide vaccine via mannose-modified LCP NPs enhanced antigen cross-presentation